Tests for long-term and short-term memory loss, judgement, thinking and processing of general information.Physical examination for reflexes, balance, sensory processes and other functions of the nervous system and the brain.Since the memory of the person is compromised, a close family member or friend may be involved during a consultation. A detailed medical history to check for the nature of memory loss, its progression, triggers, family history, substance abuse, accidents and medical problems like seizures, cancer or depression.Brain diseases like Alzheimer’s or dementia.Ī detailed evaluation is done to check for amnesia, and also to distinguish it from other disorders like dementia or Alzheimer’s disease.Inadequate flow of oxygen to the brain.Swelling in the brain due to infections.
When any part of the brain, especially the thalamus, hippocampus or other related structures, which are responsible for memories and emotions are affected, it results in amnesia.
False memories, i.e., memories invented but believed to be true.It is characterized by difficulty recalling past experiences and information. In this type of amnesia, there is difficulty processing and recollecting new information. The main types of amnesia also encompass the symptoms of the condition: People who suffer from amnesia are aware of themselves and their surroundings, but experience difficulty with new information. What are the main signs and symptoms of Amnesia? When it occurs due to a medical condition, the loss of memories, such as facts, experiences and information, is referred to as amnesia. It may occur due to information overload, stress, distraction or a host of other reasons. 24, 541–546 (1980).We all tend to forget, get confused or remember things incorrectly at times. Extending the duration of ACTH–induced memory reactivation in an amnesic paradigm. The Psychology of Human Learning: an Introduction (Longmans & Green, New York, 1942). Attenuation of emotional and nonemotional memories after their reactivation: role of beta adrenergic receptors. State-dependent recall can be induced by protein synthesis inhibition: behavioral and morphological observations. Dissociated learning in rats produced by electroconvulsive shock. Hypothermia-induced retrograde amnesia: Role of body temperature in memory retrieval. Amnesia induced by hypothermia as a function of treatment-test interval and recooling in rats. Amnesia induced by hypothermia: An unusually profound, yet reversible, memory loss. Induced recovery of memory in rats following electroconvulsive shock. Amnesia produced by electroconvulsive shock or cycloheximide: Conditions for recovery.
Teng to special treatment for amnesia trial#
The use of an extinction trial as a reminder treatment following ECS. Reinstatement of memory in rats: Dependence upon two forms of retrieval deficit following electroconvulsive shock. Reconsolidation of memory after its reactivation. Retrograde amnesia for previously acquired Pavlovian conditioning: UCS exposure as a reactivation treatment. Hypothermia-induced amnesia for newly acquired and old reactivated memories: Commonalities and distinctions. Anomalous properties of hippocampal lesion-induced retrograde amnesia. Retrieval and reconsolidation: toward a neurobiology of remembering. Amnesia: A function of the temporal relation of footshock to electroconvulsive shock. Motivational control of retrograde amnesia. Conditions that potentiate the effects of electroconvulsive shock administered 24 hours after avoidance training. Retrograde amnesia produced by electroconvulsive shock after reactivation of a consolidated memory trace: A replication. Environmental control of ECS-produced retrograde amnesia in goldfish. Retrograde amnesia produced by electroconvulsive shock after reactivation of a consolidated memory trace. The labile nature of consolidation theory. Memory involves far more than consolidation. Strengthening the shaky trace through retrieval. Fear memories require protein synthesis in the amygdala for reconsolidation after retrieval. Characteristics of retrograde amnesia following reactivation of memory in mice. Recovery of memory in chicks after disruption during learning: the reversibility of amnesia induced by protein synthesis inhibitors. Time-dependent processes in memory storage.
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